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5-Amino-1MQ

Longevity / Metabolic

An NNMT inhibitor that raises NAD+ precursors and drives fat reduction via metabolic reprogramming.

Half-life
~6–12 hours
Research dose
50–100mg/day (research)
Frequency
Daily
Routes
Oral

How it works

Inhibits Nicotinamide N-methyltransferase (NNMT) — an enzyme that consumes SAM (S-adenosylmethionine) and converts nicotinamide to 1-methylnicotinamide. By inhibiting NNMT, 5-Amino-1MQ raises SAM levels, increases the NAD+ precursor pool, boosts mitochondrial activity, and drives fat loss through metabolic reprogramming in adipose tissue.

Research uses

NAD+ precursor elevation
Fat cell metabolic reprogramming
Adipogenesis research
Obesity models
Longevity and metabolic health

Research notes

Published research from Cornell University demonstrated that 5-Amino-1MQ prevented diet-induced obesity in mice on high-fat diets and reduced adipocyte size without caloric restriction. The effect was driven primarily by NNMT inhibition in fat tissue.

Unlike direct NAD+ precursors (NR, NMN), 5-Amino-1MQ raises NAD+ indirectly by blocking its metabolic consumption. This distinguishes it mechanistically from other NAD+ support compounds.

Storage

🌡️Store at room temperature or refrigerated
🔒Keep away from moisture
📅Stable for 12+ months if stored correctly

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